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sc 03 g  (Bio-Rad)


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    Structured Review

    Bio-Rad sc 03 g
    Sc 03 G, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 246 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/sc 03 g/product/Bio-Rad
    Average 96 stars, based on 246 article reviews
    sc 03 g - by Bioz Stars, 2026-03
    96/100 stars

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    Schematic signaling pathways are involved in S1P-induced COX-2/PGE 2 expression associated with activation of caspase-3 leading to apoptosis in HCFs. S1P could be upregulated mediated through estrogen/estrogen receptor-stimulated sphingosine kinase-1 (SphK1) activity , binding with S1PR1/3 coupled to G q or G i protein results <t>in</t> <t>MMP9-mediated</t> HB-EGF cleavage leading to activation of <t>EGFR/PI3K/Akt/MAPKs</t> (i.e., p42/p44 MAPK, p38 MAPK, and JNK1/2)-dependent AP-1 c-Jun. The COX-2 transcription and PGE 2 generation are dependently regulated by an S1PR1/3-mediated EGFR transactivation to activate AP-1 binding activity. These signaling pathways contribute to the activation of AP-1 required for COX-2 expression and PGE 2 generation in HCFs.
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    sc 03 g  (Bio-Rad)
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    Schematic signaling pathways are involved in S1P-induced COX-2/PGE 2 expression associated with activation of caspase-3 leading to apoptosis in HCFs. S1P could be upregulated mediated through estrogen/estrogen receptor-stimulated sphingosine kinase-1 (SphK1) activity , binding with S1PR1/3 coupled to G q or G i protein results <t>in</t> <t>MMP9-mediated</t> HB-EGF cleavage leading to activation of <t>EGFR/PI3K/Akt/MAPKs</t> (i.e., p42/p44 MAPK, p38 MAPK, and JNK1/2)-dependent AP-1 c-Jun. The COX-2 transcription and PGE 2 generation are dependently regulated by an S1PR1/3-mediated EGFR transactivation to activate AP-1 binding activity. These signaling pathways contribute to the activation of AP-1 required for COX-2 expression and PGE 2 generation in HCFs.
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    Schematic signaling pathways are involved in S1P-induced COX-2/PGE 2 expression associated with activation of caspase-3 leading to apoptosis in HCFs. S1P could be upregulated mediated through estrogen/estrogen receptor-stimulated sphingosine kinase-1 (SphK1) activity , binding with S1PR1/3 coupled to G q or G i protein results in MMP9-mediated HB-EGF cleavage leading to activation of EGFR/PI3K/Akt/MAPKs (i.e., p42/p44 MAPK, p38 MAPK, and JNK1/2)-dependent AP-1 c-Jun. The COX-2 transcription and PGE 2 generation are dependently regulated by an S1PR1/3-mediated EGFR transactivation to activate AP-1 binding activity. These signaling pathways contribute to the activation of AP-1 required for COX-2 expression and PGE 2 generation in HCFs.

    Journal: Oxidative Medicine and Cellular Longevity

    Article Title: Sphingosine 1-Phosphate-Upregulated COX-2/PGE 2 System Contributes to Human Cardiac Fibroblast Apoptosis: Involvement of MMP-9-Dependent Transactivation of EGFR Cascade

    doi: 10.1155/2022/7664290

    Figure Lengend Snippet: Schematic signaling pathways are involved in S1P-induced COX-2/PGE 2 expression associated with activation of caspase-3 leading to apoptosis in HCFs. S1P could be upregulated mediated through estrogen/estrogen receptor-stimulated sphingosine kinase-1 (SphK1) activity , binding with S1PR1/3 coupled to G q or G i protein results in MMP9-mediated HB-EGF cleavage leading to activation of EGFR/PI3K/Akt/MAPKs (i.e., p42/p44 MAPK, p38 MAPK, and JNK1/2)-dependent AP-1 c-Jun. The COX-2 transcription and PGE 2 generation are dependently regulated by an S1PR1/3-mediated EGFR transactivation to activate AP-1 binding activity. These signaling pathways contribute to the activation of AP-1 required for COX-2 expression and PGE 2 generation in HCFs.

    Article Snippet: EGFR (sc-03-G), MMP9 (sc-10737), Akt (sc-1619), caspase-3 (sc-22140), and phospho-c-Jun (sc-822, Ser 63 ) antibodies were from Santa Cruz (Santa Cruz, CA, USA).

    Techniques: Protein-Protein interactions, Expressing, Activation Assay, Activity Assay, Binding Assay